Neurology®
The most widely read and highly cited peer-reviewed Neurology journal
Quick Search
Advanced Search
This Article
Right arrow Figures Only
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowRequest Permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Krolak–Salmon, P.
Right arrow Articles by Vighetto, A.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Krolak–Salmon, P.
Right arrow Articles by Vighetto, A.
Related Collections
Right arrow MRI
Right arrow All Cognitive Disorders/Dementia
Right arrow Infarction

Neurology 2002;58:1689-1691
© 2002 American Academy of Neurology


Brief Communications

Thalamic venous infarction as a cause of subacute dementia

P. Krolak–Salmon, MD, A. Montavont, MD, M. Hermier, MD, M. Milliery, MD and A. Vighetto, MD

From the Departments of Neurology C (Drs. Krolak–Salmon, Montavont, and Vighetto), Neuroradiology (Dr. Hermier), and Neuropsychology (Dr. Milliery), P. Wertheimer Neurological Hospital, Lyon, France.

Address correspondence and reprint requests to Dr. P. Krolak–Salmon, Service de Neurologie C, Hôpital Neurologique et Neuro-chirurgical P. Wertheimer, 59 Boulevard Pinel, 69394 Lyon Cedex 03, France.

The clinical picture of deep cerebral vein thromboses (DCVT) usually is acute, combining vigilance disorders, headaches, and focal neurologic deficit. The authors describe a patient who presented with isolated subacute dementia as the sole manifestation of DCVT. In the setting of subacute cognitive deficit, the diagnosis of DCVT must be considered when neuroimaging shows bilateral thalamic changes. Enhanced venous MR angiography is the noninvasive method of choice to ascertain the diagnosis.




This article has been cited by other articles:


Home page
NeurologyHome page
E. Carrera and J. Bogousslavsky
The thalamus and behavior: Effects of anatomically distinct strokes
Neurology, June 27, 2006; 66(12): 1817 - 1823.
[Abstract] [Full Text] [PDF]