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NEUROLOGY 1998;51:1210-1213
© 1998 American Academy of Neurology

Nervous system dysfunction in Waldenström's macroglobulinemia: Response to treatment

S. A. Rudnicki, MD, S. I. Harik, MD, M. Dhodapkar, MD, B. Barlogie, MD, PhD and D. Eidelberg, MD

From the Departments of Neurology (Drs. Rudnicki and Harik) and Medicine (Drs. Dhodapkar and Barlogie), University of Arkansas College of Medicine, Little Rock, AR; and Department of Neurology (Dr. Eidelberg), North Shore University Hospital, Manhasset, NY.

Address correspondence and reprint requests to Dr. Stacy A. Rudnicki, UAMS Department of Neurology, 4301 W. Markham, Slot 500, Little Rock, AR 72205.

A patient presented with a peripheral neuropathy and was found to have Waldenström's macroglobulinemia with high serum titers of antibodies to myelin-associated glycoprotein. He developed parkinsonism that was poorly responsive to levodopa. He failed conventional therapy and was treated with autologous bone marrow transplantation, which resulted in improvement of the neuropathy but not his parkinsonism. Critical cytoreduction in the B-cell clone may be necessary for improvement of the neuropathy of Waldenström's macroglobulinemia.

Supported in part by grant CA 55819 from the National Institutes of Health.

Received October 6, 1997. Accepted in final form June 6, 1998.




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