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Departments of Neurology (Dr. Halperin) and Pathology (Dr. Golightly), State University of New York at Stony Brook, Stony Brook, NY.
Lyme borreliosis (LB) causes a range of neurologic manifestations, the most common of which is facial nerve paralysis. To evaluate nervous system LB, we organized a neurologic collaborative study group in Suffolk County, NY, a region of high LB incidence. Between July and September 1989, LB serologies were performed on all patients with new-onset Bell's palsy. Seven of 32 had serologic evidence of LB at onset. One, initially seronegative, was highly seropositive 5 weeks later. In the five in whom we examined CSF, there was no evidence of intrathecal synthesis of specific antibody. In highly endemic areas, LB may be responsible for 1/4 of cases of Bell's palsy. Rarely, the palsy may occur prior to the development of a measurable antibody response, indicating a need for follow-up serologic testing.
Address correspondence and reprint requests to Dr. John J. Halperin, Department of Neurology, North Shore University Hospital, 300 Community Drive, Manhasset, NY 11030.
Supported in part by grants from New York State for Lyme disease research and the EEL and WHEEL Associations.
Presented in part at the 42nd annual meeting of the American Academy of Neurology, Miami Beach, FL, May 1990.
Received October 15, 1991. Accepted for publication in final form December 2, 1991.
*M. Andriola, MD; A. Belman, MD; N. Carnevale, MD, PhD; P. Carvajal, MD; M. Chacko, MD; N. Chernik, MD; P. Coyle, MD; S. deLanerolle, MD; J. Greenwood, MD; M. Gudesblatt, MD; G. Kaplan, MD, PhD; R. Moore, MD, PhD; H. Moreta, MD; F. Mortati, MD; G. Newman, MD, PhD; R. Pearl, MD; P. Poole, MD; H. Reiser, MD; A. Rosen, MD; M. Rudansky, MD; H. Sachs, PhD. MD; M. Sauter, MD; G. Schroeter, MD; J. Winter, MD; M. Zuckerman, MD.
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