Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


This Article
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Magistris, M. R.
Right arrow Articles by Roth, G.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Magistris, M. R.
Right arrow Articles by Roth, G.
NEUROLOGY 1985;35:1639
© 1985 American Academy of Neurology

Long-lasting conduction block in hereditary neuropathy with liability to pressure palsies

Michel R. Magistris, MD and Gérard Roth, MD

From the Electromyographic Unit, Department of Physical Medicine and Rehabilitation, University Hospital, Geneva, Switzerland.

We detected 29 conduction blocks in 12 patients with hereditary neuropathy and liability to pressure palsies. The blocks occurred at entrapment sites, most often the ulnar nerve at the elbow. Some had lasted for several years. This kind of block seems to be characteristic of the neuropathy, and may be related to the localized thickening of myelin ("tomacula"). In one patient with two permanent and severe blocks, neurolysis was beneficial.

Address correspondence and reprint requests to Dr. Magistris, Unité d'EMG, Hôpital Beau-Séjour, CH-1211 Geneva 4, Switzerland.

Accepted for publication February 20, 1985.




This article has been cited by other articles:


Home page
J. Neurosci.Home page
K. Adlkofer, R. Frei, D. H.-H. Neuberg, J. Zielasek, K. V. Toyka, and U. Suter
Heterozygous Peripheral Myelin Protein 22-Deficient Mice Are Affected by a Progressive Demyelinating Tomaculous Neuropathy
J. Neurosci., June 15, 1997; 17(12): 4662 - 4671.
[Abstract] [Full Text] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1985 by AAN Enterprises, Inc.